Alcoholic Cardiomyopathy: Causes, Symptoms, and Diagnosis

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They also have not identified the minimum length of time someone needs to drink alcohol before developing the condition. In our patient, acute myocardial injury and cardiomyopathy improved significantly with abstinence from alcohol. Also, low to moderate daily alcohol intake was proved to be a predictor of better prognosis for both ischemic cardiomyopathy and heart failure regardless of the presence of coronary disease[1,2]. Regarding ICD and CRT implantation, the same criteria as in DCM are used in ACM, although it is known that excessive alcohol intake is specifically linked to ventricular arrhythmia and sudden cardiac death[71].

  • The trace amounts of arsenic have not been comparable to the arsenic-in-beer endemic in Manchester but may still reach up to 10-times the amount admitted for arsenic in drinking water in the European Union and the US.
  • Pulverized antimony was used as eye shadow by Egyptian women and named al-Kol.
  • To our knowledge, our study determined prognostic factors for ACM outcome in the largest cohort of ACM patients described to date.
  • In spite of the high prevalence of excessive alcohol consumption and of its consideration as one of the main causes of DCM, only a small number of studies have analysed the long-term natural history of ACM.
  • The existence of a direct causal link between excessive alcohol consumption and the development of DCM is a controversial issue.
  • Alcohol abuse coinciding with myocarditis was reported in 1902 by McKenzie [26].
  • This activity describes the pathophysiology of ACM, its causes, presentation and the role of the interprofessional team in its management.

Of the 56 patients included in the study, 28 were former drinkers and 28 continued consuming alcohol during the study. Absorption levels of Indium-111 were high in 75% of patients who continued drinking and in only 32% of those who had withdrawn from consuming alcohol. Although some studies have detailed structural and functional damage in proportion to the amount of alcohol consumed during a patient’s lifetime[24], a large majority of authors have discarded this theory[21-23,25]. Both the absence of a direct correlation and the theory of the existence of a threshold dose (above which some alcoholics develop ACM) require the presence of individual susceptibility to alcohol induced cardiac damage[63]. It is unknown whether individual susceptibility would be related to increased vulnerability at the myocardial level and/or to impaired alcohol metabolism. Specifically, there was no evidence of a preceding viral infection or presence of another toxin.

Clinical work-up for alcoholic cardiomyopathy

In an echocardiographic study of 13 patients with alcohol-induced cardiomyopathy, five demonstrated the normalization of left ventricular function after total abstinence for six months (6). Chronic alcohol consumption can cause multi-organ damage including myocardial dysfunction. There are no specific targeted histological or immunological biomarkers for the diagnosis of alcohol-induced cardiomyopathy. Various pathophysiological mechanisms have been postulated in the development of cardiomyopathy however one key factor undergoing active research is the role of genetic mutation and susceptibility to develop cardiomyopathy. Alcoholic cardiomyopathy (ACM) is a disease in which the long-term consumption of alcohol leads to heart failure.[1] ACM is a type of dilated cardiomyopathy. “Alcoholic cardiomyopathy is a disease of the heart muscle, caused by the toxic effects of excessive alcohol consumption,” explains Professor Nik Patel, cardiologist and chairman of the academic board, Royal Society of Medicine.

alcoholic cardiomyopathy recovery time

However, very few studies have investigated the relationship between clinical characteristics and prognosis in ACM. In all ACM studies, inclusion of patients is based on patients’ self-reported alcohol drinking habits, which may lead to an underestimation of the prevalence of ACM together with problematic identification of patients who abstain and those who continue drinking. Furthermore, in many of these reports, comorbid conditions, especially myocarditis and other addictions such as cocaine and nicotine, were not reported. Ballester specifically analysed the effects of alcohol withdrawal on the myocardium using antimyosin antibodies labelled with Indium-111[72]. This radiotracer has been acknowledged as an indicator of irreversible myocardial damage.

How can I prevent this condition or reduce my risk?

The status of all patients was followed up by telephone interview, outpatient clinic attendance, or hospitalization during the follow-up period. This study protocol was approved by the Ethics Commission of Fuwai Hospital. Despite the key clinical importance of alcohol as a cause of DCM, little information has been published on the long-term outcome of patients with ACM in China. The aims of the present study were to define the long-term outcome of ACM, to compare the patient characteristics between the death and survival groups, and to determine prognostic markers.

Recovery from any mental health disorder is significantly enhanced if an individual does not drink alcohol. The rate and quality of recovery from a mental health disorder depends on a number of factors, including the person’s motivation, their engagement in treatment, their social support, etc. In some cases, especially those that are more severe, heart failure symptoms and related conditions may develop or get worse. Your healthcare provider is the best person to explain the risks and possible complications that you might face from this condition itself, related health concerns or any of the treatments that you will receive. In many — if not most — cases, abstaining from alcohol can be enough to help people recover from alcohol-induced cardiomyopathy. In cases where people don’t recover fully by abstaining from alcohol, most people will still see noticeable improvements in their symptoms.